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  • [\'Department of Internal Medicine, College of Medicine Phoenix, University of Arizona, Phoenix, AZ, USA.\', \'Division of Translational and Regenerative Medicine, Department of Medicine, College of Medicine Tucson, University of Arizona, Tucson, AZ, USA.\', \'Department of Medicine at Broward Health Medical Center, Fort Lauderdale, Florida, USA; Vascular Biology Center, Augusta University, Augusta, GA, Georgia.\', \'Vascular Biology Center, Augusta University, Augusta, GA, Georgia; Department of Anesthesiology and Perioperative Medicine and Division of Molecular and Translational Biomedicine, School of Medicine, University of Alabama at Birmingham, Birmingham, AL, USA.\', \'Division of Translational and Regenerative Medicine, Department of Medicine, College of Medicine Tucson, University of Arizona, Tucson, AZ, USA. Electronic address: steveblack@email.arizona.edu.\']
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  • -1
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?:doi
?:doi
  • S2213-2317(20)30999-X10.1016/j.redox.2020.101794
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  • Redox biology
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  • 33248422
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  • 2.203
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is ?:relation_isRelatedTo_publication of
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  • RAC1 nitration at Y32 IS involved in the endothelial barrier disruption associated with lipopolysaccharide-mediated acute lung injury.
?:type
?:year
  • 2021

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