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is ?:annotates of
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  • [\'Department of Biochemistry, Microbiology, and Immunology, Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.\', \'Faculty of Applied Medical Sciences, Taibah University, Medina, Kingdom of Saudi Arabia.\', \'Apoptosis Research Center, Children\'s Hospital of Eastern Ontario Research Institute, Ottawa, Ontario, Canada.\', \'Department of Biology, Carleton University, Ottawa, Ontario, Canada.\', \'Chronic Disease Program, Ottawa Hospital Research Institute, Ottawa, Ontario, Canada.\', \'Department of Medicine, Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.\', \'Center for Infection, Immunity, and Inflammation, Faculty of Medicine, University of Ottawa, Ontario, Canada; and.\', \'Department of Biochemistry, Microbiology, and Immunology, Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada; akumar@uottawa.ca.\', \'Department of Pathology and Laboratory Medicine, Faculty of Medicine, University of Ottawa, Ottawa, Ontario, Canada.\']
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  • -1
?:creator
?:doi
  • 10.4049/jimmunol.2001191
?:doi
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?:journal
  • Journal of immunology (Baltimore, Md. : 1950)
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?:pmid
  • 34551966
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  • -1.0
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  • -1
is ?:relation_isRelatedTo_publication of
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  • TLR-4 Agonist Induces IFN-γ Production Selectively in Proinflammatory Human M1 Macrophages through the PI3K-mTOR- and JNK-MAPK-Activated p70S6K Pathway.
?:type
?:year
  • 2021

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