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We examine the plausibility, scope and risks of aerial transmission of pathogens (including the SARS-CoV-2 virus) through respiratory droplets carried by exhaled e-cigarette aerosol (ECA). Observational and laboratory data suggests considering cigarette smoking and mouth breathing through a mouthpiece as convenient proxies to infer the respiratory mechanics and droplets sizes and their rate of emission that should result from vaping. We model exhaled ECA flow as an intermittent turbulent jet evolving into an unstable puff, estimating for low intensity vaping (practiced by 80-90% of vapers) ECA expirations the emission of 2-230 respiratory submicron droplets per puff a horizontal distance spread of 1-2 meters, with intense vaping possibly carrying hundreds and up to 1000 droplets per puff in the submicron range a distance spread over 2 meters. Bystanders exposed to low intensity expirations from an infectious vaper in indoor spaces (home and restaurant scenarios) face a 1% increase of risk with respect to a \'\'control case\'\' scenario defined by exclusively rest breathing without vaping. This relative added risk becomes 5-17% for high intensity vaping, 40-90% and over 200% for speaking or coughing (without vaping). We estimate that disinfectant properties of glycols in ECA are unlikely to act efficiently on pathogens carried by vaping expirations under realistic conditions.
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?:doi
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10.1101/2020.11.21.20235283
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document_parses/pdf_json/f811d33d8b0e9629b8f0e7e99236bc1dac7b97cd.json
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Aerial transmission of SARS-CoV-2 virus (and pathogens in general) through environmental e-cigarette aerosol
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