?:abstract
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Kidney transplant recipients are at increased risk of infection, including COVID-19, given ongoing immunosuppression. In individuals with COVID-19, complications including thrombosis and endothelial dysfunction, portend worse outcomes. In this report, we describe a kidney transplant recipient who developed severe thrombotic microangiopathy (TMA) with low platelet count (12 x 109 /L), anemia (7.5g/dL with 7% schistocytes on peripheral blood smear) and severe acute kidney injury concurrent with COVID-19. The clinical course improved following plasma exchange. Given this presentation, we hypothesize that COVID-19 triggered TMA.
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