PropertyValue
?:abstract
  • There is pressing urgency to understand the pathogenesis of the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) which causes Coronavirus disease 2019 (COVID-19). The tissue tropism of SARS-CoV-2 includes not only the lung but also the vascular and integumentary systems. Angiotensin-converting enzyme 2 (ACE2) appears to be the key functional receptor for the virus. There is a prominent innate immune response to SARS-CoV-2 infection, including inflammatory cytokines, chemokines, the complement system, and acute phase proteins. The pathophysiologic significance of SARS-COV-2 and host immune system interaction, and COVID-19-associated coagulopathy instigating microvascular injury syndrome mediated by activation of complement pathways, and an associated procoagulant state is important for wound care professionals to understand.
?:creator
?:journal
  • Int_Wound_J
?:license
  • unk
?:publication_isRelatedTo_Disease
?:source
  • WHO
?:title
  • Pathophysiology of coronavirus disease 2019 for wound care professionals
?:type
?:who_covidence_id
  • #798871
?:year
  • 2020

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